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By ; fh4djh4r

A wealth of information has accumulated on the subject of hypertension, and the number of publications dedicated to topics in this field of study continues to increase. It has become nearly impossible for medical professionals to absorb all of the diverse information and to gather the information into a coherent theoretical concept and practical approach to treating the disease, even for those of us who are actively involved in this research specialty on a daily basis. It becomes a still greater challenge for the many health care providers whose primary focus is clinical practice or for scientists who are doing research in basic science but whose work is relevant to the subject of hypertension.
Our goal for Hypertension:

Principles and Practice has been to compile and present information about the most relevant
underlying principles of hypertension and apply them to clinical practice. It takes facets of basic research and applies these concepts to bedside management. This concomitant emphasis on principles and practice confers a novel and distinctive quality to the book.
This reference is designed for use by primary care physicians, cardiologists, endocrinologists, nephrologists, vascular
specialists, hypertension specialists, pharmacologists, scientists, nurses, students, and others, who want to obtain accurate, comprehensive, and up-to-date information on all aspects of hypertension in an attractive and easily readable format. The editors, contributors, and publisher envision this book as a new international platform for up-to-date information for the interested reader.

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By : fh4djh4r

Genomics and Pharmacogenomics in Anticancer Drug Development and Clinical Response provides the most comprehensive body of knowledge available on the role of genetic and genomic variation in the individualization of drug therapies in cancer patients. As a consequence of the intrinsic chromosomal and genetic instability of the tumor genome, it is generally believed that tailoring of chemotherapy in cancer patients might be achieved by molecular analysis of patient tumor DNA. In addition, to reduce the toxicity risk of patients, the tumor DNA information should be integrated with the available data on polymorphic drug-metabolizing enzyme and transporter genes mediating the exposure of patients to active drugs and/or their active metabolites. The chapters of this book clearly show how DNA information from both the host (germline) and the tumor should be taken into account for rational selection of drug therapies in cancer patients, an aspect that received little attention, despite its importance.

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By : fh4djh4r

Oxidative stress is a major factor in health, aging, and disease and is often defined by the redox balance established by cellular oxidants and antioxidants. The ratio of reduced to oxidized levels of major low-molecular weight protein thiols, such as cysteine and glutathione or thioredoxin and glutaredoxin, is often used as a measure of oxidative stress. Among naturally occurring thiols, the discovery of a-lipoic acid has led to an unprecedented interest in basic research because of its role in energy metabolism and as an antioxidant for the treatment of certain
diseases. a-Lipoic acid has acquired importance because of its role as a coenzyme in the maintenance of energy metabolism, on one hand, and as an antioxidant and cell redox modulator, on the other hand. The former was supported by the discovery of the essential role of lipoic acid in mitochondrial dehydrogenasedriven reactions inherent in energy metabolism and the latter is supported by reduction of supplemental a-lipoic acid to dihydrolipoic acid in mammalian cells conferring further antioxidant protection. For example, lipoic acid is an essential cofactor of a-keto acid dehydrogenase complexes in eukaryotic cells as part of the lipoyl domain of the E2 enzymes (dihydrolipoamide acyltransferases). Also, exogenous R- (or RS)-lipoic acid is reduced by a flavoenzyme, dihydrolipoamide dehydrogenase, of the complex in mitochondria. Hence lipoic acid may be a mitochondrial-targeted nutrient. The cell redox status is a critical factor regulating cell proliferation, differentiation, apoptosis (programmed cell death), and necrosis, and the mitochondrion is a center of interest for its role in the generation of signaling molecules (e.g., hydrogen peroxide and nitric oxide) that, in turn, modulates the redox status of the cell.

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